Relationship between Cardiopulmonary, Mitochondrial and Autonomic Nervous System Function Improvement after an Individualised Activity Programme upon Chronic Fatigue Syndrome Patients

Figures

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  Figure 1. CONSORT-type flow diagram.

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  Figure 2. Influence of IAP on fatigue scales. (a) influence of IAP on CFS—Chronic Fatigue Scale. (b) influence of IAP on FSS—Fatigue Severity Scale. (c) influence of IAP on FIS—Fatigue Impact Scale. IAP—Individualised Activity Program, Orange dots connected by black line indicate median value, vertical black lines denote interquartile range. Blue dots before and after denote results of individual patients.

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  Figure 3. Influence of IAP on VO2peak. VO2peak mL/kg/min—maximal oxygen uptake during physical exercise measured in ml/kg/min, IAP—Individualised Activity Program, Orange dots connected by black line indicate median value, vertical black lines denote interquartile range. Blue dots before and after denote the results of individual patients.

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  Figure 4. Influence of IAP on Mitofusins. (a) influence of IAP on miofusin1 level. (b) influence of IAP on mitofusin2 level. IAP—Individualised Activity Program, Orange dots connected by black line indicate median value, vertical black lines denote interquartile range. Blue dots before and after denote the results of individual patients.

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  Figure 5. Interaction between influence of IAP on VO2peak and Mfn1. VO2peak—maximal oxygen consumption obtained during physical exercise, mitofusin1—level of mitofusin1, IAP—Individualised Activity Program.

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  Figure 6. Interaction between influence of IAP on VO2subpeak and sympathovagal balance in response to head-up tilt test. VO2AT—maximal oxygen consumption during anaerobic threshold, LF/HF-RRI_HUTT—low frequency diastolic blood pressure to high frequency R-R interval during head-up tilt test, IAP—Individualised Activity Program.