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Highlights the role of metabolic disturbances and faulty adrenergic regulation in myocardial failure, emphasizing the use of new technologies to build clinical models.
Cardiac failure can begin with metabolic disturbances within the myocardial cell--in the mitochondria, contractile proteins, or other cellular organelles. It also can result from faulty adrenergic regulation of contraction. New technology is making it possible to synthesize the contributing factors into a clinical model that will greatly enhance our understanding of these mechanisms.
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Richard J. Bing (1983) studied this question.
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