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This article presents the hypothesis that exercise-associated autonomic and neuroendocrine failure leads to increased hypoglycemia in patients with type 1 diabetes mellitus.
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This article proposes that dysfunctional autonomic regulation after exercise or hypoglycemia contributes to exercise-induced hypoglycemia in type 1 diabetes.
In Brief Type I diabetes mellitus patients who tightly control blood glucose levels toward normal have increased frequency of hypoglycemia, a major barrier to physical activity. This article presents the hypothesis that dysfunctional autonomic regulation of metabolism after recent bouts of exercise or hypoglycemia contributes to exercise-induced hypoglycemia in these patients. This article will advance the hypothesis that exercise associated autonomic and neuroendocrine failure leads to increased hypoglycemia in type 1 diabetes mellitus patients.
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Sandoval et al. (2006) conducted a review in Type 1 diabetes mellitus. Exercise-associated autonomic and neuroendocrine failure was evaluated on Exercise-induced hypoglycemia. This article presents the hypothesis that exercise-associated autonomic and neuroendocrine failure leads to increased hypoglycemia in patients with type 1 diabetes mellitus.
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