Why the study?
Aging is an independent risk factor for cardiovascular diseases, but the mechanisms of cardiomyocyte senescence and cellular communications within myocardial microenvironments require synthesis.
Design
Review
Authors
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Highlights senescence pathways in cardiac aging; leaves open whether targeting them prevents remodeling or failure in older adults.
Key points are not available for this paper at this time.
Tang et al. (2020) studied this question.
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