Population
31 subjects (12 patients with congestive heart failure and 19 healthy subjects)
Design
Cross-sectional
Authors
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Preserved baroreflex and respiratory modulation of MSNA in CHF; leaves open non-baroreflex drivers of sympathetic overactivity for targeted investigation.
Modulation of MSNA by the arterial baroreflex and respiration is preserved in CHF, suggesting that higher sympathetic nerve firing rates in CHF are caused by factors other than the loss of regulation by these inhibitory influences.
Ando et al. (1997) studied this question.
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