Key result
Treatment with heart failure medications, vitamin D, and calcium led to full clinical, biochemical, and echocardiographic recovery in a 6-month-old infant with rachitic hypocalcemic cardiomyopathy.
Case Report (n=1)
Hypocalcemic cardiomyopathy secondary to rickets is a life-threatening but reversible condition in infants that requires prompt recognition and treatment with calcium and vitamin D.
Prompt recognition may enable reversal of hypocalcemic cardiomyopathy in rachitic infants; leaves open confirmation in larger studies.
Cardiomyopathy in infants is characterized by heart failure in apparently normal children without previous organic cardiac lesions. Cardiomyopathy has been found to comprise four types. Rickets is common in Saudi Arabia, that is why I reviewed this subject. Recently this case with classical features of rickets being admitted in a serious state we thought of publishing it. The infant responded well to treatment and full recovery was achieved. Follow up biochemistry, radiology cardiac function completely recovered and bony abnormalities showed evidence of healing. This case might have been missed as respiratory infection. We recommend meticulous look for biochemical features of rickets in infants admitted with respiratory symptoms.
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Elidrissy et al. (2016) conducted a case report in Rachitic hypocalcemic cardiomyopathy (n=1). Medical therapy (digoxin, captopril, phenobarbitone, vitamin D, calcium iv, iron, magnesium) was evaluated on Clinical, biochemical, and echocardiographic recovery. Treatment with heart failure medications, vitamin D, and calcium led to full clinical, biochemical, and echocardiographic recovery in a 6-month-old infant with rachitic hypocalcemic cardiomyopathy.
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