Key result
Xiaoyao San significantly ameliorated high-fat diet-induced hepatic steatosis and liver injury in NAFLD rats by regulating the cAMP/PKA-mediated PPARα, FOXO1, and NF-κB signaling pathways.
Why the study?
There is no approved pharmaceutical intervention for NAFLD, and while Xiaoyao San has shown therapeutic efficacy, its underlying mechanisms remain unclear.
Does Xiaoyao San improve nonalcoholic fatty liver disease in a high-fat diet-induced rat model?
Population
High-fat diet-induced NAFLD rat model
Comparison
Xiaoyao San vs untreated or control conditions
Design
Preclinical animal study with transcriptomics and proteomics analysis
Authors
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Hypothesis-generating for Xiaoyao San in rat NAFLD; leaves open human translation pending clinical trials.
Does Xiaoyao San improve nonalcoholic fatty liver disease in a high-fat diet-induced rat model?
Xiaoyao San improves nonalcoholic fatty liver disease in a rat model by regulating the cAMP/PKA-mediated PPARα, FOXO1, and NF-κB signaling pathways.
Liu et al. (2025) studied Nonalcoholic Fatty Liver Disease (NAFLD) (n=24). Xiaoyao San (XYS) vs. Normal saline was evaluated. Xiaoyao San significantly ameliorated high-fat diet-induced hepatic steatosis and liver injury in NAFLD rats by regulating the cAMP/PKA-mediated PPARα, FOXO1, and NF-κB signaling pathways.
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