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February 19, 2005Journal of General Virology

Intersegmental recombination between the haemagglutinin and matrix genes was responsible for the emergence of a highly pathogenic H7N3 avian influenza virus in British Columbia

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Authors

JPJohn PasickCanadian Food Inspection Agency KHKatherine HandelCanadian Food Inspection Agency JRJohn H. RobinsonNewcastle University

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Implication

Indicates potential for rapid LPAI-to-HPAI shifts in poultry; leaves open insertion mechanisms and surveillance needs.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Pasick et al. (2005) studied this question.

synapsesocial.com/papers/6a887af5c16cbaca05f4d6df https://doi.org/10.1099/vir.0.80478-0

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Also Consider

Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context:

1Heterogeneity in the haemagglutinin gene and emergence of the highly pathogenic phenotype among recent H5N2 avian influenza viruses from Mexico1996 · 274 citations
2RNA VIRUS MUTATIONS AND FITNESS FOR SURVIVAL1997 · 1,547 citations
3Attenuation of pathogenicity of fowl plague virus by recombination with other influenza A viruses nonpathogenic for fowl: nonexculsive dependence of pathogenicity on hemagglutinin and neuraminidase of the virus1976 · 83 citations