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Angiotensin II effects on rat hypothalamic catecholamines warrant no clinical extrapolation; leaves open central mechanisms in human cardiovascular regulation.
Intravenous administration of angiotensin II reduced the adrenaline content, increased the catechol-O-methyltransferase activity, and decreased the monoamine oxidase activity of rat hypothalamus. 2. Intraventricularly administrated angiotensin II reduced the noradreanline content to a smaller extent. 3. The change produced by intravenous administration of angiotensin might be indirectly caused by a response to angiotensin.
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Iijima et al. (1976) studied this question.
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