Key result
Post-myocardial infarction leukocyte diversity and specialized proresolving mediators are critical in determining resolving versus nonresolving inflammation and subsequent cardiac remodeling.
Design
Review
Authors
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Highlights novel therapeutic targets to prevent adverse post-MI remodeling; leaves open whether modulating these inflammatory.
Understanding the mechanisms of resolving and nonresolving inflammation driven by leukocyte diversity post-myocardial infarction may help identify novel therapeutic targets to prevent adverse cardiac remodeling and heart failure.
Tourki et al. (2017) conducted a review in Myocardial infarction. Leukocyte diversity was evaluated. Post-myocardial infarction leukocyte diversity and specialized proresolving mediators are critical in determining resolving versus nonresolving inflammation and subsequent cardiac remodeling.
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