Why the study?
Data were needed on the prevalence, clinical profile, risk factors, and prognosis of elevated Troponin I in SVT among non-CAD patients.
What is the prevalence of elevated Troponin I in patients with SVT and what clinical variables are associated with this elevation?
What is the prevalence of elevated Troponin I in patients with SVT and what clinical variables are associated with this elevation?
Elevated Troponin I occurs in 36% of patients presenting with SVT and is associated with older age, longer tachycardia duration, and higher heart rate, but does not necessarily indicate obstructive coronary artery disease.
Troponin elevation in SVT warrants caution against assuming ACS; leaves open need for prospective studies on mechanisms and outcomes.
Cardiac troponins are the major sensitive and specific markers of myocardial injury. Joint European Society of Cardiology/American College of Cardiology committee has given the redefinition of myocardial infarction (MI) stating that troponins are the preferred cardiac markers for detecting myocardial injury. Besides MI, elevated troponins are also seen in tachyarrhythmias, acute cerebrovascular accident, acute pulmonary thromboembolism, chronic kidney disease, septic shock, myocardial injury. Though Troponin I frequently increases during myocardial injury, the etiology may vary from coronary to non- coronary causes. Paroxysmal supraventricular tachycardia (PSVT) is the commonest form of arrhythmia seen in the Emergency Department. In contrast to ventricular tachyarrhythmias these patients are haemodynamically stable and they have good prognosis on treatment. Serum cardiac troponin I (cTnI) testing provides momentous risk stratification in these group of arrhythmias. This study will provide comprehensive data on prevalence, clinical profile, risk factors and prognosis of patients with elevated Troponin I in SVT in non CAD patients.
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Kiruthika et al. (2021) studied this question.
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