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AF pathophysiology hypotheses remain diverse; leaves open identification of dominant pathways for targeted therapies.
Pathomechanisms underlying atrial fibrillation (AF) are very complex and include various electrical and structural remodelling processes involving focal ectopic activity of atrial cells and reentry mechanisms through atrial tissue. This article reviews basic mechanisms related to AF and gives an update on hypotheses, which are currently thought to provide the most comprehensive concepts.
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Namdar et al. (2014) studied this question.
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