Key result
CaMKII inhibition has emerged as a highly validated molecular mechanism and potential broad-spectrum antiarrhythmic strategy for treating cardiac arrhythmias.
Design
Review
Authors
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Supports CaMKII inhibition as antiarrhythmic target; leaves open validation in randomized trials.
The multifunctional Ca2+/calmodulin-dependent protein kinase II (CaMKII) emerges as a highly validated molecular mechanism and potential broad-spectrum antiarrhythmic therapeutic target.
Rokita et al. (2012) conducted a review in Cardiac arrhythmias. CaMKII inhibition was evaluated. CaMKII inhibition has emerged as a highly validated molecular mechanism and potential broad-spectrum antiarrhythmic strategy for treating cardiac arrhythmias.
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