Key result
This review summarizes preclinical evidence on the role of beta-adrenergic receptors and estrogen in regulating the inflammatory response and cardiac repair after myocardial infarction.
Why the study?
Deciphering the pathophysiological mechanisms underlying post-MI cardiac repair is critical to favorably regulate cardiac wound repair and prevent heart failure.
This review highlights the pathophysiological mechanisms by which catecholamines and estrogen regulate cardiac repair and remodeling after myocardial infarction in preclinical models.
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Preclinical signals on beta-adrenergic and estrogen effects post-MI remain hypothesis-generating; leaves open any clinical translation.
Matarrese et al. (2021) conducted a review in Myocardial Infarction. β-adrenergic receptors and estrogen was evaluated. This review summarizes preclinical evidence on the role of beta-adrenergic receptors and estrogen in regulating the inflammatory response and cardiac repair after myocardial infarction.
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