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sequence of changes was found during rehabilitation. The patterns were of prognostic value in some children electrocardiographic abnormalities persisted after recovery, as judged by clinical and laboratory criteria. Cardiac arrest was suspected as a cause of sudden death in kwashiorkor. This may have been related to fluid, protein, and electrolyte shifts in the presence of a wasted and weakened heart. Attention is drawn to certain similarities between the heart in kwashiorkor and the myocardiopathy of unknown origin of the adult Bantu. An interesting finding was histological changes in the heart muscle indistinguishable from those described in the adult form of heart disease.
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Walt et al. (1962) studied this question.
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