Key result
TNFAIP3 and TP53INP2 showed significantly higher expression and protein secretion levels in AMI patients compared to healthy individuals.
Why the study?
Programmed cell death plays a crucial role in the occurrence and development of acute myocardial infarction, prompting the exploration of new biomarkers and therapeutic targets.
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Hypothesis-generating for PCD biomarkers in AMI; leaves open validation before any clinical application.
Sun et al. (2023) studied Acute myocardial infarction. Acute myocardial infarction vs. Healthy controls was evaluated on Differentially expressed genes related to programmed cell death. TNFAIP3 and TP53INP2 showed significantly higher expression and protein secretion levels in AMI patients compared to healthy individuals.
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