Transient T wave inversion following paroxysmal tachycardia does not invariably indicate serious organic heart disease, and prognosis relies heavily on the patient's overall clinical condition.
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Transient T-wave inversion after tachycardia warrants interpretation in clinical context rather than as isolated pathology; leaves open prospective validation of prognostic factors.
Graybiel and White (1934) have stressed that care must be' taken in the diagnosis of serious heart disease when inverted T1 or T2 waves are the only abnormal finding, and that this inversion is not invariably associated with organic disease. They cited two cases of paroxysmal tachycardia in young robust adults, one followed by inverted T1 and the other by inverted T2 and T3 waves; there was no sign of organic disease and there was a gradual return to a normal cardiogram.-Campbell and Elliot (1939) record two cases of ventricular tachycardia that gave transient changes simulating coronary thrombosis; one of these gave a history of diphtheria and the other was of long standing and had been suffering from increasing dyspncea during the intervals between attacks; both died suddenly at a later date. These cases show that the general condition must be given prime consideration in forming a prognosis.
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Geoffrey Currie (1942) studied this question.
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