A 1932 case report discussing the clinical differentiation of ventricular paroxysmal tachycardia from supraventricular origins.
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Early differentiation criteria warrant cautious application today; leaves open validation against current electrophysiologic standards.
Article1 June 1932Ventricular Paroxysmal Tachycardia With Report of a CaseEDWARD W. HOLLINGSWORTH, M.D., F.A.C.P.EDWARD W. HOLLINGSWORTH, M.D., F.A.C.P.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-5-12-1506 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptParoxysmal tachycardia may be defined as an abnormally rapid heart action with sudden onset and offset, and is divided according to the site of origin of the impulse which may be in the auricle, auriculo-ventricular node, or ventricle.In general, the important point is to differentiate those examples of ventricular origin from others inasmuch as in them the prognosis is grave. There are several points of distinction clinically between auricular or supra-ventricular, and ventricular paroxysms, the former having a practically constant rate in the various attacks and being frequently stopped by vagal pressure,1especially left, the latter varying in rate...References1 COHNFRASER AEFR: Paroxysmal tachycardia and the effect of stimulation of the vagus nerves by pressure, Heart, 1913-14, v, 93-108. Google Scholar2 STRONGLEVINE GFSA: The irregularity of the ventricular rate in paroxysmal ventricular tachycardia, Heart, 1923, x, 125-131. Google Scholar3 LEVINE SA: The clinical recognition of paroxysmal ventricular tachycardia, Am. Heart Jr., 1927, iii, 177-179. CrossrefGoogle Scholar4 SCHWENSEN C: Ventricular tachycardia as the result of the administration of digitalis, Heart, 1921-1922, ix, 199-205. Google Scholar5 LEVY RL: Clinical studies of quinidine. II. Alterations in the cardiac mechanism after administration of quinidine to patients with auricular fibrillation with consideration of certain toxic effects of the drug, Arch. Int. Med., 1922, xxx, 541-477. (Levine11 and Fulton13 have criticized the author's interpretation of his curves apparently with justification.) Google Scholar6 ROBINSONHERRMANN GCGR: Paroxysmal tachycardia of ventricular origin and its relation to coronary occlusion, Heart, 1921, viii, 59-81. Google Scholar7 LEWIS T: The experimental production of paroxysmal tachycardia and the effects of ligation of the coronary arteries, Heart, 1909-1910, i, 98-137. Google Scholar8 LEWIS T: The mechanism and graphic registration of the heart beat, 1925. Shaw & Sons, London, 255. Google Scholar9 SCOTT RW: Observations on a case of ventricular tachycardia, with retrograde conduction, Heart, 1922, ix, 297-309. Google Scholar10 STRAUSS MB: Paroxysmal ventricular tachycardia, Am. Jr. Med. Sci., 1930, clxxix, 337-345. CrossrefGoogle Scholar11 LEVINE SA: Clinical electrocardiography, Oxford Medicine, Oxford University Press, 370 (37). Google Scholar12 BARNESWHITTEN ARMB: Study of the R-T interval in myocardial infarction, Am. Heart Jr., 1929, v, 142-171. CrossrefGoogle Scholar13 LEVINEFULTON SAMN: The effect of quinidine sulphate on ventricular tachycardia; clinical observations, Jr. Am. Med. Assoc., 1929, xcii, 1,162-1,168. CrossrefGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: Veterans Administration Hines, Illinois*Published with the permission of the Medical Director, Veterans Administration, who assumes no responsibility for the opinions expressed or the conclusions drawn by the writer.†Received for publication, September 3, 1931. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 1 June 1932Volume 5, Issue 12Page: 1506-1512KeywordsHeartTachycardia ePublished: 1 December 2008 Issue Published: 1 June 1932 PDF downloadLoading ...
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Edward W. Hollingsworth (1932) studied this question.
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