Why the study?
Does an anti-rod monoclonal antibody inhibit smooth muscle myosin's activity and assembly?
Does an anti-rod monoclonal antibody inhibit smooth muscle myosin's activity and assembly?
This study demonstrates that smooth muscle myosin's activity is affected by its state of assembly, with filamentous structure stimulating the activity of individual myosin molecules.
May influence smooth muscle contractility via assembly state; leaves open relevance to human cardiovascular disease.
Monoclonal antibodies specific for the rod region can affect smooth muscle myosin's motor properties. Actin movement by phosphorylated myosin was inhibited by an antibody (LMM.4) which binds to the COOH-terminal end of the coiled-coil rod, a region thought to be involved in filament assembly. The actin-activated ATPase activity of the myosin-antibody LMM.4 complex was also reduced 10-fold at actin concentrations that gave maximal turnover rates with filamentous myosin. Metal-shadowing of the phosphorylated myosin-antibody complex at low ionic strength showed small bundles of parallel extended molecules, instead of filaments. Five other anti-rod antibodies had little or no effect on myosin's ability to act as a motor. This is the first demonstration that a muscle myosin's activity is affected by its state of assembly. A common theme that emerges from the studies on both muscle and non-muscle myosins is that assembly into a filamentous structure stimulates the activity of the individual myosin molecules.
No takes yet. Share an insight, caveat, or question.
Horowitz et al. (1992) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: