Highlights that a significant proportion of intestinal infarction cases in cardiac failure lack an ascertained vascular occlusion.
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Alerts to possible nonocclusive mesenteric ischemia in HF; leaves open mechanistic and management questions.
A REVIEW of the literature on intestinal infarction soon discloses that in a significant proportion of cases the cause of the infarction is not ascertained.1 Not infrequently the physician believes that a thrombus or embolus had actually been present but was lost or dislodged during the post-mortem examination.1 (An unfortunate but frequently convenient excuse for this mysterious disappearance is the assumed ineptness of the resident.) So often has this question arisen that a few pathologists request that the attending physicians witness the dissection of the mesenteric vessels to convince them that in some cases no vascular occlusion has in fact . . .
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Norman Ende (1958) studied this question.
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