Population
Rat atrial myocytes
Design
Preclinical
Authors
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Does not support clinical changes in arrhythmia care; leaves open purinergic modulation of atrial KACh channels in vivo.
Extracellular ATP and UTP can directly activate acetylcholine-activated K+ channels in rat atrial myocytes via G protein-coupled mechanisms.
Wu et al. (1998) studied this question.
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