Short-term (15 min) renal ischemia enhances the role of neurogenically released NPY in sympathetic vasoconstriction, whereas prolonged (2 h) ischemia reversibly reduces noradrenaline release and vascular reactivity.
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May inform mechanisms of ischemia-duration effects on renal sympathetic control; leaves open translation to human reperfusion injury.
Malmström et al. (1997) studied this question.
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