This study demonstrates significant collateral vessel development in a chronic canine model of coronary occlusion, highlighting its utility for studying coronary insufficiency and myocardial infarction.
Animal data warrant no practice change; extends canine ameroid model for coronary collateral research.
A detailed angiographic study is presented demonstrating the number and size of collateral vessels that form as the result of placing a single ameroid constrictor on the left circumflex artery of the canine heart. Different routes of collateral formations are also shown. This remarkable collateral development and the implications of using a chronic canine model for studying coronary insufficiency and myocardial infarction are outlined.
No takes yet. Share an insight, caveat, or question.
Marlon et al. (1973) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: