Population
Experimental models of kidney damage, cultured renal and vascular cells, and rodents
Design
Editorial
Authors
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Angiotensin II acts as a pro-inflammatory cytokine mediating renal and vascular injury via NF-κB activation, providing a mechanistic basis for the anti-inflammatory and tissue-protective effects of ACE inhibitors and ARBs.
Angiotensin II acts as a pro-inflammatory cytokine mediating renal and vascular injury via NF-κB activation, providing a mechanistic basis for the anti-inflammatory and tissue-protective effects of ACE inhibitors and ARBs.
Ruiz‐Ortega et al. (2000) studied this question.
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