Population
Reconstituted complex of actin, heavy-meromyosin, tropomyosin and troponin from at-death and 168 hr…
Comparison
168 hr postmortem storage vs At-death muscle (0 hr)
Design
Preclinical
Authors
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May explain postmortem ATPase changes in animal muscle; leaves open cardiac contractility implications in humans.
The increase in myofibrillar ATPase activity during postmortem storage of muscle is likely due to increased actin-myosin interaction associated with troponin degradation.
Ikeuchi et al. (1980) studied this question.
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