Why the study?
Cardiotoxicity from anticancer therapies presents an increasing clinical challenge as the population of cancer survivors grows.
Anticancer treatments such as chemotherapy and radiation therapy can cause a wide spectrum of cardiovascular complications, requiring awareness and collaboration between oncologists and cardiologists.
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May inform arrhythmia care in expanding cancer survivor population; leaves open optimal strategies pending prospective data.
Cardiovascular diseases and cancer are among the main causes of death in developed countries. Early detection of malignant pathology, the continuous development of cancer therapy and improvement in the quality of patient care makes the survival of patients in this group constantly growing. It should be noted that people who have been cured of cancer are one of the largest, constantly growing subgroups that have access to health care. Cardiovascular diseases occurring in oncological patients are often a consequence of anti-cancer treatment, but they may also result from a malignant disease. The occurrence of cardiotoxicity in cancer patients, which is the result of applied oncological treatment, is an increasing challenge for both oncologists and cardiologists. The toxic effect of anticancer treatment on the cardiovascular system may manifest itself in a wide spectrum of changes such as electrophysiological abnormalities, vascular toxicity or heart failure. In this paper, we will discuss the latest views on the cardiotoxicity associated with various chemotherapeutic agents and radiation therapy used.
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Dudek et al. (2019) studied this question.
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