Key result
Antibodies from multiple sclerosis patients cross-reacted with both myelin basic protein and Epstein-Barr virus latent membrane protein 1, suggesting molecular mimicry in MS pathogenesis.
Population
Blood lymphocytes from patients with multiple sclerosis (MS)
Design
Preclinical
Authors
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May implicate EBV in MS autoimmunity; animal data leave open human therapeutic relevance.
Antibodies induced against EBV LMP1 may cross-react with myelin basic protein, suggesting molecular mimicry as a potential mechanism for MS pathogenesis.
Gabibov et al. (2011) studied Multiple sclerosis. Antibodies from MS patients was evaluated on Cross-reactivity of scFv clones with myelin basic protein and Epstein-Barr latent membrane protein 1. Antibodies from multiple sclerosis patients cross-reacted with both myelin basic protein and Epstein-Barr virus latent membrane protein 1, suggesting molecular mimicry in MS pathogenesis.
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