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Iron Metabolism in Tuberculosis-Roberts et al. beng similar to the abnormalities produced by isoniazid in the marrows of experimental animals and in human subjects reeiving isoniazid therapy. The exact cause of the change is unknown, but it has been attributed to disturbance of pyridoxine metabolism. The incidence of the abnormality in the present series might have been higher had iron deficiency, which was common in the group as a whole, been corrected before the bone-marrow examinations were made.
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Hurdle et al. (1966) studied this question.
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