Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
October 4, 2012Journal of VirologyOpen Access

IRF-3 and IRF-7 work in concert to initiate antiviral responses that restrict murine norovirus replication in vivo and are required for induction of IFN-α/β in macrophages.

View Full Paper
Ask AI
Bookmark
Share

Population

Mice with floxed alleles of the IFNAR1 chain of the IFN-α/β receptor, cultured macrophages, and dendritic…

Design

Preclinical

Key result

IRF-3 and IRF-7 work in concert to initiate antiviral responses that restrict murine norovirus replication in vivo and are required for induction of IFN-α/β in macrophages.

Authors

LTLarissa B. ThackrayEDErning DuanHLHelen M. Lazear

Discussion

Loading...

Member takes

Overview

Animal data identify IRF-3/7 as innate immunity targets; leaves open translation to human norovirus or clinical use.

Structured PICO

P
Population
Mice with floxed alleles of the IFNAR1 chain of the IFN-α/β receptor, cultured macrophages, and dendritic cells
E
Exposure
Murine norovirus (MNV) infection and assessment of IRF-3 and IRF-7 function
O
Outcome
MNV replication in vivo and in cultured cellssurrogate

Expression of the IFN-α/β receptor on macrophages/neutrophils and dendritic cells, along with IRF-3 and IRF-7, is critical for innate immune responses to norovirus infection.

Cite This Study

Thackray et al. (2012) studied Norovirus infection. IRF-3 and IRF-7 expression was evaluated on Murine norovirus replication. IRF-3 and IRF-7 work in concert to initiate antiviral responses that restrict murine norovirus replication in vivo and are required for induction of IFN-α/β in macrophages.

synapsesocial.com/papers/6a957722c163d75667fa8ee4https://doi.org/10.1128/jvi.01824-12
View Full Paper
Ask AI
Bookmark
Share