A1- and A2-adenosine receptors and substance P receptors are present on ventricular epicardial sensory nerve endings, mediating tonic activity and increased activation during myocardial ischemia.
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Supports adenosine and substance P receptors in ischemia-induced cardiac afferent activation; hypothesis-generating for targeted modulation, requires human validation.
Huang et al. (1995) studied this question.
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