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January 1, 2020Journal of Central Nervous System DiseaseOpen Access

Endothelial Dysfunction and the Effect of Arginine and Citrulline Supplementation in Children and Adolescents With Mitochondrial Diseases

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Why the study?

Mitochondrial diseases feature nitric oxide deficiency leading to endothelial dysfunction and microvascular hypoperfusion, and the effect of supplementing nitric oxide precursors arginine and citrulline required investigation.

Does arginine or citrulline supplementation improve reactive hyperemic index in children and adolescents with mitochondrial diseases?

Population

Children and adolescents with mitochondrial diseases

Comparison

Arginine and citrulline supplementation

Key result

Arginine or citrulline supplementation increased the reactive hyperemic index, indicating improved endothelial dysfunction in children and adolescents with mitochondrial diseases.

Authors

FJFatma Al JasmiNZNuha Al ZaabiKAKhalid Al‐Thihli

Discussion

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Member takes

Overview

May support arginine or citrulline for endothelial dysfunction in mitochondrial disease; hypothesis-generating and requires randomized confirmation before practice change.

Structured PICO

Does arginine or citrulline supplementation improve reactive hyperemic index in children and adolescents with mitochondrial diseases?

P
Population
Children and adolescents with mitochondrial diseases evaluated for endothelial dysfunction and the response to arginine and citrulline supplementation.
I
Intervention
Arginine or citrulline supplementation
O
Outcome
Reactive hyperemic index (RHI) measured by peripheral arterial tonometrysurrogate

Arginine or citrulline supplementation may improve endothelial dysfunction in children and adolescents with mitochondrial diseases by enhancing nitric oxide production.

Cite This Study

Jasmi et al. (2020) studied Mitochondrial diseases. Arginine and citrulline supplementation was evaluated on Reactive hyperemic index (RHI). Arginine or citrulline supplementation increased the reactive hyperemic index, indicating improved endothelial dysfunction in children and adolescents with mitochondrial diseases.

synapsesocial.com/papers/6a95dcb6bc2accd33153956bhttps://doi.org/10.1177/1179573520909377
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