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Liver transplantation is associated with hepatocellular damage to the transplant outcome known as ischemia/reperfusion (I/R) syndrome. Despite advances in liver storage, complications associated with cold injury still limit the optimal use of transplantation as a therapeutic intervention in the event of graft failure. One of the therapeutic challenges is to develop new treatments to protect and better preserve liver tissue damaged by a sequence of ischemia/reperfusion.
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Gdara et al. (2020) studied this question.
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