described, but in our two patients there was no other obvious cause for this condition.At the time of their admission for myocardial infarction both patients had taken diamorphine, prochlorperazine, and glyceryl trinitrate; one patient (case 1) also took a few doses of frusemide and cyclopenthiazide, and the other required co-proxamol and buprenorphine intermittently over the first few days.None of these drugs, however, is known to cause peripheral neuropathy.Though we may have observed no more than a chance association, we suggest that patients who complain of weakness or lethargy after a myocardial infarct should undergo careful neurological examination.We thank Dr L D Blumhardt for arranging electrophysiological studies on these patients.
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Gaitonde et al. (1987) studied this question.
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