Answers are grounded in this paper and cite their sources.
Calcification in a renal mass is not uncommon and is usually considered to be associated with malignancy, particularly when central in location. The following case describes renal calcification resulting from infarction. A 55-year-old woman was admitted as an emergency in renal failure following a history of several days of nausea and vomiting. In the past she had had both aortic and mitral valve replacements for rheumatic heart disease and as a result was taking digoxin, frusemide and Warfarin. On admission her serum digoxin level was 4.63 ng/ml (normal range 0.8−2.0 ng/ml) and this was thought to account for her symptoms. Further investigation revealed her blood urea to be 102 m.mol/1 and potassium 6.3 m.mol/1. Serum calcium was normal. An emergency ultrasound examination was performed to exclude ureteric obstruction and she was started on peritoneal dialysis.
No takes yet. Share an insight, caveat, or question.
Hicks et al. (1984) studied this question.
Synapse has enriched 10 related papers. Consider them for comparative context: