Key result
Reovirus infection in male broilers resulted in degenerative changes in sheath and tendon fibroblasts, including cytoplasmic vacuolization and cell disruption, up to 20 weeks postinoculation.
Reovirus infection in broilers causes significant ultrastructural degenerative changes in tendon and sheath fibroblasts even in the absence of identifiable virus particles.
Indicates persistent tendon fibroblast damage after reovirus in broilers; leaves open translation to other species or clinical settings.
Gastrocnemius tendon and sheath from male broilers infected with reovirus at 1 day of age were evaluated ultrastructurally at 1, 2, 3, 4, 5, 10, 15, and 20 weeks postinoculation (PI). Although virus particles were not identified, degenerative changes in sheath and tendon fibroblasts were characterized by cytoplasmic vacuolization, disruption of membranes, and loss of ribosomes from rough endoplasmic reticulum, mitochondrial degeneration, and cell disruption.
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Hill et al. (1989) studied Reovirus infection. Reovirus infection was evaluated on Ultrastructural changes in gastrocnemius tendon and sheath. Reovirus infection in male broilers resulted in degenerative changes in sheath and tendon fibroblasts, including cytoplasmic vacuolization and cell disruption, up to 20 weeks postinoculation.
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