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March 13, 2022Journal of Cellular and Molecular MedicineOpen Access

ANGPTL4 regulate glutamine metabolism and fatty acid oxidation in nonsmall cell lung cancer cells

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Why the study?

ANGPTL4 is highly expressed in various cancers, but its regulation of energy metabolism in tumours remains to be determined.

Does ANGPTL4 regulate energy metabolism and proliferation in nonsmall cell lung cancer cells?

Population

Two NSCLC cell lines (A549 and H1299) and mice

Comparison

ANGPTL4 knockdown or overexpression vs controls

Design

Preclinical laboratory and animal study

Key result

Knockdown of ANGPTL4 inhibited energy metabolism, glutamine consumption, fatty acid oxidation, and proliferation in NSCLC cells, as well as tumor metastasis in mice.

Authors

XSXiao SongWNWang Nai‐dongLTLong Tian

Discussion

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Member takes

Overview

May identify ANGPTL4 as NSCLC metabolic target; hypothesis-generating in animal models and should not change practice.

Structured PICO

Does ANGPTL4 regulate energy metabolism and proliferation in nonsmall cell lung cancer cells?

P
Population
Nonsmall cell lung cancer (NSCLC) cell lines (A549 and H1299) and a mouse model
I
Intervention
Knockdown or overexpression of ANGPTL4, glutamine deprivation, and treatment with GLS and CPT1 inhibitors
C
Comparator
Normal NSCLC cells or control conditions
O
Outcome
Energy metabolism (aerobic glycolysis, glutamine consumption, fatty acid oxidation) and cell proliferation/tumour metastasissurrogate

ANGPTL4 promotes glutamine consumption and fatty acid oxidation, driving energy metabolism and proliferation in nonsmall cell lung cancer.

Cite This Study

Song et al. (2022) studied Non-small cell lung cancer (NSCLC). ANGPTL4 knockdown and overexpression vs. Control/normal NSCLC cells was evaluated on Energy metabolism, glutamine consumption, fatty acid oxidation, and cell proliferation. Knockdown of ANGPTL4 inhibited energy metabolism, glutamine consumption, fatty acid oxidation, and proliferation in NSCLC cells, as well as tumor metastasis in mice.

synapsesocial.com/papers/6a98662de63c287d3d9fb0c9https://doi.org/10.1111/jcmm.16879
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