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A 4-year-old girl had tick paralysis initially with astasia-abasia, areflexia, and nystagmus. Motor weakness appeared as a second phase of her illness and all symptoms cleared with removal of the tick. The toxin, experimentally, appears to have various sites of action in the neuromuscular system. This correlates with the clinical course of this patient and others reported in the literature.
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Jerome S. Haller (1972) studied this question.
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