Key result
A 75-year-old woman developed reversible left ventricular apical ballooning (neurogenic stunned myocardium) associated with a postictal catecholamine surge following status epilepticus.
Case Report (n=1)
No
Patients suffering from intractable seizures may harbor a risk of postictal catecholamine surge and catecholamine-induced myocardial dysfunction, presenting as neurogenic stunned myocardium.
Postictal cardiac dysfunction warrants consideration after status epilepticus; single case leaves open need for prospective studies on neurogenic stunning.
A 75-year-old woman developed left ventricular apical ballooning, shortly after recovering from status epileptics. Plasma noradrenaline and adrenaline levels were 2.05 ng/ml and 0.48 ng/ml, respectively. Endomyocardial biopsy disclosed patchy areas of interstitial myocardial fibrosis, atrophy and vacuolization of cardiac myocytes, and some disappearance of myocyte nuclei. Follow-up echocardiography showed that the left ventricular apical ballooning was restored to normal within 25 days. These findings are compatible with neurogenic stunned myocardium. It is important to recognize that patients suffering from intractable seizures may harbor a risk of postictal catecholamine surge and catecholamine-induced myocardial dysfunction.
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Shimizu et al. (2008) conducted a case report in Neurogenic stunned myocardium (n=1). Status epilepticus and postictal catecholamine surge was evaluated. A 75-year-old woman developed reversible left ventricular apical ballooning (neurogenic stunned myocardium) associated with a postictal catecholamine surge following status epilepticus.
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