In an animal model of type 2 diabetes, insulin resistance triggers intestinal machinery to oversecrete apo B-48-lipoproteins, contributing to hypertriglyceridemia.
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Should not change clinical lipid management in diabetes; hypothesis-generating for intestinal apoB-48 overproduction as a driver of hypertriglyceridemia.
Zoltowska et al. (2003) studied this question.
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