Key result
A 786 km ultra-trail race linked to a ~5-fold CK increase without acute kidney damage.
Case Report (n=1)
Answers are grounded in this paper and cite their sources.
Extreme ultra-trail racing can induce transient exertional rhabdomyolysis without necessarily causing acute kidney injury or hyponatremia.
A case study involving a healthy trained male athlete who completed a 786 km multi-stage ultra-trail race. Several markers were analyzed in blood and urine samples: creatinine (SCR) for kidney damage, sodium ([Na+]) for hyponatremia, creatine kinase (CK) for exertional rhabdomyolysis, as well as other hematological values. Samples were taken before and after the race and during the recovery period (days 2 and 9 after the race). Results showed: SCR = 1.13 mg/dl, [Na+] =139 mmol/l and CK = 1.099 UI/l. Criteria for the determination of acute kidney damage were not met, and [Na+] concentration was above 135 mEq/L, indicating the absence of hyponatremia. Exertional rhabdomyolysis was suffered by the athlete (baseline CK increased fivefold), though this situation was reverted after 9 days of recovery. Ultra-trail races cause biochemical changes in athletes, which should be known about by healthcare professionals.
No takes yet. Share an insight, caveat, or question.
Lecina et al. (2021) conducted a case report in Healthy trained male athlete (n=1). 786 km multi-stage ultra-trail race vs. Baseline (before the race) was evaluated on Markers of kidney damage (SCR), hyponatremia ([Na+]), and exertional rhabdomyolysis (CK). Completion of a 786 km ultra-trail race induced exertional rhabdomyolysis with a fivefold increase in baseline CK, but did not cause acute kidney damage or hyponatremia.
Synapse has enriched 10 related papers. Consider them for comparative context: