Key result
Hyperemesis-induced hypomagnesemia and hypokalemia caused ventricular tachycardia in a pregnant patient with a structurally normal heart.
Case Report (n=1)
Highlights the rare occurrence of ventricular tachycardia due to severe electrolyte imbalance (hypomagnesemia and hypokalemia) from hyperemesis gravidarum in a patient with a structurally normal heart.
Alerts clinicians to electrolyte-induced VT risk in hyperemesis gravidarum; leaves open need for prospective prevention studies.
Hyperemesis gravidarum is persistent vomiting, seen more often in the first trimester of pregnancy, when the patient is unable to maintain adequate hydration. Intractable vomiting can lead to severe electrolyte imbalance, which may cause electrocardiogram abnormalities. Occasionally, ventricular tachycardia can complicate a pregnancy. Although its occurrence usually indicates an underlying cardiac structural or arrhythmic abnormality, it may rarely occur in a pregnant patient with structurally normal heart. We report a rare case of ventricular tachycardia, secondary to hyperemesis induced hypomagnesemia and hypokalemia, in a pregnant patient with a structurally normal heart.
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Kochhar et al. (2018) conducted a case report in Hyperemesis Gravidarum (n=1). Hyperemesis-induced hypomagnesemia and hypokalemia was evaluated. Hyperemesis-induced hypomagnesemia and hypokalemia caused ventricular tachycardia in a pregnant patient with a structurally normal heart.
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