Population
Anaesthetised dog preparation following transection of the cervical vagi and in the presence of a…
Design
Preclinical
Key result
Inhibition of neuronally released NO with TRIM reduced bradycardia evoked by preganglionic but not postganglionic vagal stimulation, indicating NO facilitates vagal control at parasympathetic ganglia.
Authors
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NO facilitation at cardiac ganglia may guide autonomic research; leaves open translation to human vagal therapies.
This study demonstrates that neuronally released nitric oxide facilitates vagal control of heart rate primarily at the preganglionic-postganglionic synaptic level in cardiac parasympathetic ganglia.
Markos et al. (2002) studied this question. 1-(2-trifluoromethylphenyl) imidazole (TRIM) was evaluated on Bradycardia resulting from stimulation of preganglionic and postganglionic parasympathetic fibres. Inhibition of neuronally released NO with TRIM reduced bradycardia evoked by preganglionic but not postganglionic vagal stimulation, indicating NO facilitates vagal control at parasympathetic ganglia.
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