Case Report (n=1)
Paroxysmal sympathetic hyperactivity is a rare but important complication of hypoxic brain injury following cardiac arrest that can be managed with targeted pharmacotherapy such as clonidine and diazepam.
May support adjunctive clonidine and diazepam for refractory PSH post-arrest; hypothesis-generating and requires prospective validation.
Paroxysmal sympathetic hyperactivity (PSH) is a syndrome of an increased sympathetic drive after brain injury. PSH has been previously referred with multiple different names. It is seen most commonly after a traumatic brain injury, but rarely it has been reported after infections, brain malignancies, and brain injury after cardiac arrest. We present a case of a young male who developed PSH after cardiac arrest and will discuss clinical features and various management options.
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Singh et al. (2018) conducted a case report in Paroxysmal Sympathetic Hyperactivity (PSH) after cardiac arrest (n=1). Clonidine and diazepam was evaluated on Symptom control.
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