Why the study?
Paroxysmal sympathetic hyperactivity (PSH) can occur after stroke but is often misdiagnosed, leading to delayed treatment and prolonged hospital stay.
Case Report (n=1)
No
Highlights the presentation and management of paroxysmal sympathetic hyperactivity syndrome following recurrent stroke in an elderly patient.
Hypothesis-generating for post-stroke PSH management; prospective trials needed before clinical adoption.
Paroxysmal sympathetic hyperactivity (PSH) is a state of autonomic dysfunction characterized by symptoms such as tachypnea, tachycardia, hypertension, hyperthermia, sweating, and dystonia. It can occur after traumatic brain injury, hypoxic-ischemic encephalopathy, and diseases such as stroke. Hypoxia, extensive axonal damage, and young age are believed to predispose to the development of PSH. These patients may be diagnosed with pulmonary embolism, septicemia, or epileptic seizures. Delays in diagnosis prolong hospital stay. Here, we present an 81-year-old man who developed PSH after a recurrent stroke and our management.
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Bilgili et al. (2024) conducted a case report in Paroxysmal sympathetic hyperactivity after recurrent stroke (n=1). Propranolol, trazodone, and gabapentin was evaluated on Frequency and severity of sympathetic attacks.
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