Key result
Oxygen breathing at 8 to 12 L/min increased mean Po2 from 58-72 mm Hg to 106-160 mm Hg and achieved >95% hemoglobin saturation in 34 patients with acute myocardial infarction.
Why the study?
Does oxygen breathing improve arterial hypoxemia in patients with acute myocardial infarction?
Observational (n=34)
Does oxygen breathing improve arterial hypoxemia in patients with acute myocardial infarction?
Arterial hypoxemia in acute myocardial infarction is likely due to elevated pulmonary venous pressure causing a similar initial pulmonary defect across varying degrees of clinical severity, with a consistent 11% true right-to-left shunt.
No takes yet. Share an insight, caveat, or question.
Oxygen corrects hypoxemia in AMI; hypothesis-generating for routine use, should not yet change practice.
Sukumalchantra et al. (1970) conducted an observational in Acute myocardial infarction (n=34). Oxygen breathing vs. Baseline (before oxygen breathing) was evaluated on Arterial blood gases (Po2) and hemoglobin saturation. Oxygen breathing at 8 to 12 L/min increased mean Po2 from 58-72 mm Hg to 106-160 mm Hg and achieved >95% hemoglobin saturation in 34 patients with acute myocardial infarction.
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