Population
Human non-small cell lung cancer cell lines (A549 and H1299) and male nude mice xenograft models
Comparison
Hypoxia exposure, ANGPTL4 overexpression or… vs Normoxia, control vectors, and no irradiation
Design
Preclinical
Follow-up
up to 30 days for xenograft model
Key result
Hypoxia-induced ANGPTL4 promoted radioresistance in non-small cell lung cancer through intracellular and exosomal pathways by inhibiting GPX4-dependent ferroptosis.
Authors
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May support ANGPTL4-ferroptosis targeting to overcome hypoxic radioresistance in NSCLC; leaves open human translation.
Hypoxia-induced ANGPTL4 promotes radioresistance in non-small cell lung cancer by inhibiting ferroptosis, highlighting a potential therapeutic target to improve radiotherapy efficacy.
Zhang et al. (2022) studied Non-small cell lung cancer. ANGPTL4 modulation and exosomal transfer vs. Control (normoxia or negative control vectors) was evaluated. Hypoxia-induced ANGPTL4 promoted radioresistance in non-small cell lung cancer through intracellular and exosomal pathways by inhibiting GPX4-dependent ferroptosis.
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