Key result
Monounsaturated fatty acids release ~78% of LPL from endothelial cell surfaces via direct interaction.
The amount and type of fatty acids may regulate lipoprotein lipase activity in vivo by affecting its interaction with glycosaminoglycans and apolipoprotein C-II.
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May modulate endothelial LPL availability in vivo; leaves open dietary fatty acid effects on human triglyceride metabolism.
Saxena et al. (1990) studied this question. Fatty acids was evaluated on Release of LPL from endothelial cell surfaces. Monounsaturated, polyunsaturated, and saturated fatty acids released 78%, 60%, and 28% of LPL from endothelial cell surfaces, respectively, by interacting directly with LPL.
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