Key result
High energy cardioversion (24 J) via an implantable cardioverter defibrillator reproducibly initiated a new ventricular tachycardia, whereas low energy (3 J) successfully terminated the original VT.
Why the study?
Does high energy cardioversion via ICD induce new ventricular tachycardia in a patient with drug-refractory monomorphic VT?
Case Report (n=1)
Does high energy cardioversion via ICD induce new ventricular tachycardia in a patient with drug-refractory monomorphic VT?
High energy cardioversion from an ICD can paradoxically induce new ventricular tachycardias, highlighting a potential proarrhythmic effect.
High-energy ICD shocks may induce secondary VT; this case report leaves open whether energy titration improves device programming.
A transvenous implantable cardioverter defibrillator (ICD) was implanted into a 58 year old woman with idiopathic dilated cardiomyopathy who had drug refractory monomorphic ventricular tachycardia (VT). Antitachycardia pacing failed to terminate the VT; termination was attempted at 24 J, which was above the defibrillation threshold. When cardioversion at 24 J was delivered, VT with a different morphology and slower rate was reproducibly initiated. At 3 J, however, the original VT was successfully terminated without initiation of the slower VT. A new VT may be induced by high energy cardioversion. This may be a manifestation of the proarrhythmic potential of ICDs.
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Chinushi et al. (1997) conducted a case report in Idiopathic dilated cardiomyopathy with drug refractory monomorphic ventricular tachycardia (n=1). High energy cardioversion via ICD vs. Low energy cardioversion (3 J) was evaluated on Initiation of new ventricular tachycardia. High energy cardioversion (24 J) via an implantable cardioverter defibrillator reproducibly initiated a new ventricular tachycardia, whereas low energy (3 J) successfully terminated the original VT.
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