Key result
Infant and environmental stress modify vital cardiocirculatory, respiratory, and arousal controls, potentially contributing to SIDS.
SIDS may result from a combination of immature cardiorespiratory controls and cumulative environmental or infant stressors.
Hypothesis-generating for SIDS prevention; leaves open causal validation before any practice change.
Preventable risk factors for sudden infant death syndrome include the infant’s environment and response characteristics. Various infant and environmental factors thus modify the vital cardiocirculatory, respiratory and arousal controls in healthy infants. Similar changes in cardiorespiratory and autoresuscitative responses have been found in the analysis of sleep recordings of victims of sudden infant death syndrome. It is not known why some infants die, while others show similar changes but survive in the first year of life. The death could be due to the degree of the initial immature controls, to the severity of the additional challenge, or to a combined effect of inadequate autoresuscitative mechanisms and the cumulative influence of infant and/or environmental stressors.
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Kahn et al. (2015) conducted a review in Sudden infant death syndrome. Infant and environmental stress factors was evaluated. Infant and environmental stress factors modify vital cardiocirculatory, respiratory, and arousal controls, potentially contributing to sudden infant death syndrome.
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