Key result
Neurocirculatory asthenia linked to temporary Lead II T-wave inversions in two healthy young women.
Case Report (n=2)
No
Temporary T-wave inversion in Lead II can occur in patients with neurocirculatory asthenia without organic heart disease.
Transient Lead II T-wave inversion may be benign in neurocirculatory asthenia; leaves open generalizability beyond these cases.
Article1 May 1944INVERSION OF THE T-WAVES OF THE ELECTROCARDIOGRAM IN TWO PATIENTS WITH NEUROCIRCULATORY ASTHENIAWEBSTER MERRITT, M.D., F.A.C.P.WEBSTER MERRITT, M.D., F.A.C.P.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-20-5-773 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptUntil a few years ago inversion of the T-waves in Lead I or II of the electrocardiogram was considered evidence of organic heart disease. Recently, however, certain exceptions to this general rule have been reported. At Riverside Hospital during the past five years, we have studied two young women who apparently do not have heart disease but whose electrocardiograms have shown temporary inversion of the T-waves in Lead II. We are presenting their case histories after a brief review of the literature.Graybiel and White,1in 1935, reported seven young people, three male and four female, suffering with neurocirculatory asthenia,...Bibliography1. GRAYBIELWHITE APD: Inversion of the T-wave in Lead I or II of the electrocardiogram in young individuals with neurocirculatory asthenia, with thyrotoxicosis, in relation to certain infections, and following paroxysmal ventricular tachycardia, Am. Heart Jr., 1935, x, 345. CrossrefGoogle Scholar2. BREEDFAULKNER WBJM: Errors in the clinical application of electrocardiography, ANN. INT. MED., 1936, x, 58. Google Scholar3. AKESSON S: Ueber Veränderungen des Elektrokardiogramms bei orthostatischer Zirkulationsstörung, Upsala Läkaref. Förh., 1936, xli, 383. Google Scholar4. SIGLER LH: Electrocardiographic changes occurring with alterations of posture from recumbent to standing positions, Am. Heart Jr., 1938, xv, 146. CrossrefGoogle Scholar5. GRAYBIELWHITE APD a. : Electrocardiography in practice, 1941, W. B. Saunders, Philadelphia, p. 27. b. Ibid., p. 25. c. Ibid., p. 161. d. Ibid., p. 157. Google Scholar6. GRAYBIEL A: Personal communication. Google Scholar7. GRAYBIELSTARRWHITE ARSPD: Electrocardiographic changes following the inhalation of tobacco smoke, Am. Heart Jr., 1938, xv, 89. CrossrefGoogle Scholar8. SEGAL HL: Cigarette smoking: Effect on the electrocardiogram with and without the use of filters, Am. Jr. Med. Sci., 1938, cxcvi, 851. CrossrefGoogle Scholar9. STEARNSDRINKERSHAUGHNESSY WHCKTJ: The electrocardiographic changes found in 22 cases of carbon monoxide (illuminating gas) poisoning, Am. Heart Jr., 1938, xv, 434. CrossrefGoogle Scholar10. BARKERSHRADERRONZONI PSELE: The effect of alkalosis and of acidosis upon the human electrocardiogram, Am. Heart Jr., 1939, xvii, 169. CrossrefGoogle Scholar11. GREENEGILBERT CANC: Studies on the responses of the circulation to low O2 tension, Arch. Int. Med., 1921, xxvii, 517. CrossrefGoogle Scholar12. KATZHAMBURGERSCHUTZ LNWWWJ: The effect of generalized anoxemia on the electrocardiogram of normal subjects, Am. Heart Jr., 1934, ix, 771. CrossrefGoogle Scholar13. LEVYBRUENNRUSSELL RLHGNG: The use of electrocardiographic changes caused by induced anoxemia as a test for coronary insufficiency, Am. Jr. Med. Sci., 1939, cxcvii, 241. CrossrefGoogle Scholar14. MAINZERKRAUSE FM: Changes of the electrocardiogram brought about by fear, Cardiologia, 1939, iii, 286. CrossrefGoogle Scholar15. TUTTLEKORNS WWHM: Electrocardiographic observations on athletes before and after a season of physical training, Am. Heart Jr., 1941, xxi, 104. CrossrefGoogle Scholar16. WHITE PD: Heart disease, Second Edition, 1937, The Macmillan Co., New York, p. 140. Google Scholar17. LAFFITTE LS: Personal communication. Google Scholar18. DAMESHEKLOMANMYERSON WJA: Human autonomic pharmacology VII. The effect on the normal cardiovascular system of acetyl-beta-methyl-choline chloride, atropine, prostigmin, benzedrine—with especial reference to the electrocardiogram, Am. Jr. Med. Sci., 1938, cxcv, 88. CrossrefGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: Jacksonville, Florida*Delivered before the American College of Physicians Section of the Florida State Medical Society, Jacksonville, April 28, 1941. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byFunctional Heart DiseaseStudies concerning the etiology and pathogenesis of neurocirculatory asthenia 1 May 1944Volume 20, Issue 5Page: 773-778KeywordsCardiovascular therapyElectrocardiography ePublished: 1 December 2008 Issue Published: 1 May 1944 PDF downloadLoading ...
No takes yet. Share an insight, caveat, or question.
Webster Merritt (1944) conducted a case report in Neurocirculatory asthenia (n=2). Neurocirculatory asthenia was evaluated on Temporary inversion of T-waves in Lead II. Two young women without apparent heart disease presented with temporary inversion of T-waves in Lead II associated with neurocirculatory asthenia.
Synapse has enriched one closely related paper. Consider it for comparative context: